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HCG is not recommended for use in females and would not normally be required as within bodybuilding it is typically utilized in order to recommence testosterone production. In fact, if LH levels were at normal (i.e., within the normal ranges, then the presence of a low circulating concentration of LH can cause symptoms) one would therefore think that the presence of an elevated LH should have been sufficient to cause symptoms of hypogonadism. If, on the other hand, the concentrations are above normal (i.e., > 10 ug/l in females, i.e., > 300 ug/l in males), then the use of the GnRH agonist may have caused a condition which was not treated with testosterone and the patient has been placed on GnRH agonists for treatment. For example, we previously noted that one GnRH agonist alone (testolizumab) provided a 30% increase in the mean amplitude of the LH peak in hyperandrogenic males at 4 months, and the amplitude was further increased to a 70% increase at 8 months, a 120% increase at 12 months, and a 180% increase at 16 months. Similarly, one of the first GnRH agonists studied for treatment of the patient who suffered from hyperandrogenism was an injection of testosterone which did provide a symptom-free increase in LH. This drug is also indicated for patients with other hypothalamic and pituitary diseases like adrenal fatigue and hypogonadism. A second condition that may be found in those who experience a hyperandrogenic effect on their sex lives is hypothalamic amenorrhea. When women experience symptoms that are not typically the result of endogenous (i.e., testosterone) production, this may be due to low levels of GnRH agonist in the plasma. For example, one of the most common causes of amenorrhea is a condition known as HSDD, or high levels of SHBG in men. According to the American Society of Clinical Endocrinology (ASCEND), "a woman's LH-to-FSH ratio may be low if she has a high SHBG concentration. This condition should be evaluated by a clinician in the early stages and managed by taking oral GnRH agonist(s)" (American Society for Clinical Endocrinology 2000). These patients can be managed with medication or with GnRH agonist injections (see "Hormonal Therapy for Hyperandrogenism"). If one wants to go into more detail on the hypothalamic effects of the GnRH agonist, the human pituitary tumor of pituitary 1 has been studied extensively. Pituit Similar articles: